Toggle light / dark theme

MMTP Campaign update “Aging is a disease”.

Crowdfunding Campaign: https://www.lifespan.io/campaigns/the-major-mouse-testing-program/

We are testing a combination of compounds which clear out dysfunctional cells in the body, called Senolytics, to see if we can extend maximum lifespan and healthspan in mice. Please subscribe, share, and fund our Lifespan.io campaign today!

MMTP — Major Mouse Testing Program.

Interesting read on Chemo’s impact on the brain and brain functioning known as Chemo Brain.


During and after chemotherapy, many cancer patients describe feeling a mental fog, a condition that has been dubbed “chemo brain.” Why this happens is unclear, but researchers have found a new clue to understanding this syndrome. A study in the journal ACS Chemical Neuroscience reports that chemotherapy in rats affects their chemical messengers dopamine and serotonin, which are associated with cognition.

Read more

Purely tragic. Hopefully, doctors without borders or experts can assist this child.


A baby boy’s head has swollen to more than three times its normal size due to a medical condition.

Two-year-old Emon, from southern Bangladesh, has a head that weighs 20 lbs (9kg) — the same weight as an average one-year-old boy.

3D Stem Cells — definitely makes sense given the organ, skin graffe, etc. produce on 3D printers in today’s labs.


JERUSALEM – Israeli 3D printer firm Nano Dimension has successfully lab-tested a 3D bioprinter for stem cells, paving the way for the potential printing of large tissues and organs, the company said on Wednesday.

While 3D printers are used already to create stem cells for research, Nano Dimension said the trial, conducted with Israeli biotech firm Accellta Ltd, showed its adapted printer could make large volumes of high resolution cells quickly.

“3D bioprinting enabled by the two companies’ technologies, means that Nano Dimension and Accellta have the potential to accelerate high fidelity and high viability manufacturing of living cellular products,” the companies said.

Treatment with sex hormones recovers serious genetic diseases cells, this is the first demonstration that the lengthening of telomeres is possible in humans with the use of a medication,” says the researcher.


Estudo demostrou que há como estimular a enzima telomerase por meio de hormônios sexuais, tanto masculinos quanto femininos.

Por — Editorias: Ciências, Ciências Biológicas, Ciências da Saúde.

Read more

Engineers at the University of California San Diego have developed the first flexible wearable device capable of monitoring both biochemical and electric signals in the human body. The Chem-Phys patch records electrocardiogram (EKG) heart signals and tracks levels of lactate, a biochemical that is a marker of physical effort, in real time. The device can be worn on the chest and communicates wirelessly with a smartphone, smart watch or laptop. It could have a wide range of applications, from athletes monitoring their workouts to physicians monitoring patients with heart disease.

Nanoengineers and electrical engineers at the UC San Diego Center for Wearable Sensors worked together to build the device, which includes a flexible suite of sensors and a small electronic board. The device also can transmit the data from biochemical and electrical signals via Bluetooth.

Nanoengineering professor Joseph Wang and electrical engineering professor Patrick Mercier at the UC San Diego Jacobs School of Engineering led the project, with Wang’s team working on the patch’s sensors and chemistry, while Mercier’s team worked on the electronics and data transmission. They describe the Chem-Phys patch in the May 23 issue of Nature Communications.

Read more

May turn out to be the “fountain-of-youth gene,” say researchers.


An atherosclerotic lesion. Such lesions can rupture and cause heart attacks and strokes. (credit: UVA School of Medicine)

University of Virginia School of Medicine have discovered that a gene called Oct4 — which scientific dogma insists is inactive in adults — actually plays a vital role in preventing ruptured atherosclerotic plaques inside blood vessels, the underlying cause of most heart attacks and strokes.

The researchers found that Oct4 controls the conversion of smooth muscle cells into protective fibrous “caps” inside plaques, making the plaques less likely to rupture. They also discovered that the gene promotes many changes in gene expression that are beneficial in stabilizing the plaques. In addition, the researchers believe it may be possible to develop drugs or other therapeutic agents that target the Oct4 pathway as a way to reduce the incidence of heart attacks or stroke.